Variable amounts of cobalt are absorbed, and apparently quickly excreted in the urine. Unabsorbed cobalt is lost in feces.
A dietary deficiency of cobalt, per se has not been produced in humans. Its significant and role in human nutrition, as far as is known is specified to its presence in vitamin B12. It is essential in the formation of red blood cells.
In animals, if the feed is deficient in cobalt, severe anemia occurs. But deficiency of cobalt is not common among human beings.
For example , cattle and sheep in certain regions develop a peculiar disease characterized by emaciation and anemia.
Cobalt ion is present in cobamide, a derivative of vitamin B12 and one of the most extraordinary of all biologically active substance. Cobamide is important in the synthesis of amino acids used to make proteins. Deficiency of cobalt can lead to fatal disorder pernicious anemia.
Deficiency of Cobalt
